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Now You See Them, Now You Don't: The Amyloid Channel Hypothesis

Ever since amyloid-β (Aβ) was identified as the major component in amyloid plaques, scientists have been trying to decipher just exactly what the little peptide does. Most researchers would agree that Aβ is toxic to cells, but some are still skeptical. ...

ADCS Prevention Instrument Project

There is now a concerted push in the research community to find pharmacological and/or non-pharmacological interventions that can prevent or slow the progression of Alzheimer disease. But prevention trials promise to be long and potentially costly affairs ...

How Cognitive Stimulation Might Protect Against Aβ Oligomers

Cognitive stimulation has been shown to lower the risk of Alzheimer’s disease, but what actually happens in the brain to confer protection? In the March 6 Neuron, researchers led by Dennis Selkoe report that β2-adrenergic signaling plays a key role. Wild ...

BDNF and Alzheimer's Disease—What's the Connection?

Nicole C. Berchtold and Carl W. Cotman led this live discussion on 25 November 2003. Readers are invited to submit additional comments by using our Comments form at the bottom of the page. Transcript: Nicole C. Berchtold and Carl W. Cotman led this live ...

Vascular Factors in Alzheimer's Disease

Jack de la Torre, David Bennett, and Julie Schneider led this live discussion on 21 April 2004. Readers are invited to submit additional comments by using our Comments form at the bottom of the page. Transcript: This live discussion with Jack de la Torre ...

Protein Misfolding: An Unfolding Series of Discussions

Dominic Walsh led this live discussion on 9 June 2004. Readers are invited to submit additional comments by using our Comments form at the bottom of the page. Transcript: Dominic Walsh (University College Dublin) led this live discussion on 9 June 2004. ...

Webinar: Cortex Aging Too Fast? Blame TMEM106B and Progranulin

If age is the strongest risk factor for Alzheimer’s and other neurodegenerative diseases, how come some nonagenarians stay sharp while others lose their edge? Genetics beyond the known genes linked to pathologies, such as amyloid plaques and ...

Slow Aβ Clearance Is ApoE4’s Modus Operandi in Late-Onset AD

Alzforum has been pleased to partner with the journal Science Translational Medicine in hosting a Webinar discussion of a new Alzheimer’s disease research study published 29 June 2011. The journal generously granted free access to Castellano et al. for ...

Evolution of AD Trials

In a sign that researchers are grappling with therapy development, the 4th International Conference on Clinical Trials on Alzheimer's Disease was filled beyond the capacity of its venue, drawing 522 researchers from around the globe. Held 3-5 ...

Stress Granules and Neurodegenerative Disease—What’s the Scoop?

The formation of cytoplasmic inclusions called stress granules is one of nature’s many ways of coping with adverse situations. It appears that these inclusions sequester non-essential messenger RNAs, allowing cells to focus on making proteins, such as ...

Adding Dynamics and Nuance to Alzheimer’s Staging

Being able to image amyloid, glucose metabolism, atrophy, even network connectivity in living patients has been a boon for those trying to stage the progression of Alzheimer’s disease. Using magnetic resonance imaging (MRI) and positron emission ...

Learn About AD Biomarker Meta-Analysis on AlzBiomarker Database

In the April 8 Lancet Neurology, researchers led by Bob Olsson at the University of Gothenburg, Sweden, reported the most comprehensive meta-analysis of published Alzheimer’s disease fluid biomarker data to date. The researchers scoured literature going ...

Have a topic idea for a webinar? We would love to hear it. Send an email to webinars [at] alzforum [dot] org.

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