Is Alzheimer’s Disease a Uniquely Human Disorder?
participant is whether there are protective sites that are differentially phosphorylated in different species
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Webinar: Pathogenic Protein Spread? Let’s Think Again
aggregation of α-synuclein in enteric or other peripheral sites before emergence of synuclein toxicity in the
Fluid Business: Could “Liquid” Protein Herald Neurodegeneration?
localizes to sites of transcription in cells, they hypothesized it might interact with RNA polymerase. Sure
Computational Modeling—Will it Rescue AD Clinical Trials?
The Alzheimer's field has had its share of clinical trial flops. Now there's a push to learn from past failures. Computational scientists in pharmaceutical companies are guiding clinical trial design by first putting drug candidates through thei
Leaky Blood-Brain Barrier a Harbinger of Alzheimer's?
To sate the brain’s voracious hunger for energy, thousands of capillaries deliver blood and oxygen to every sulcus and gyrus, and the white and gray matter in between. Herein lies a problem. The delicate cells of the brain must be protected from the flots
New Frontier: Developing Outcome Measures for Pre-dementia Trials
other (due to entry criteria, patient population differences, and study site differences) that the data
Using ADNI Data—Online Training, Part 1
visualization tools available on the ADNI data site. A second training session to be scheduled at a later date
Neuroinflammation—A Prelude to Alzheimer's?
models, there is a substantial increase in APP expression and accumulation at the sites of injury,
Who Should Use the New Diagnostic Guidelines? The Debate Continues
out appropriately from tertiary into secondary and primary care sites throughout the community? Has
Resolving Controversies on the Path to AD Therapeutics
Disease—Time for Bright New Ideas?, Dr. Herrup at Rutgers on Slide 8 stated that “Site of plaques ≠ site of
Intraneuronal Aβ: Was It APP All Along?
On 16 June 2011, ARF held a Webinar with Eddie Lee, Todd Golde, Gunnar Gouras, Frank LaFerla, Virginia Lee, Lars Nilsson, and Robert Vassar to discuss this study’s implications for intraneuronal Aβ and its role in AD pathogenesis. The Guest Moderator was
Grasping the Shadow Force: Immune Cells in ALS
bloodstream to swarm in and mingle with microglia—the nervous system’s resident immune cells—around sites of
Reimagining Alzheimer's Disease—Time for Bright New Ideas?
Alois Alzheimer made a major breakthrough when he discovered senile plaques in the brains of dementia patients, and his work ultimately led to the discovery of amyloid-β and to the amyloid cascade hypothesis. But that cascade is only part of a much bigger
Herpes Simplex and Alzheimer’s—Time to Think Again?
site. As Letteneur explained, it is not possible to routinely sample CSF, which might give a better
Microdissection and Microarrays: Analyzing Selective Cell Vulnerability
As Tolstoy wrote in Anna Karenina, “Happy families are all alike; every unhappy family is unhappy in its own way.” The same might be said of the brain. Normal brains look and behave similarly, but those affected by different neurodegenerative diseases are
Have a topic idea for a webinar? We would love to hear it. Send an email to webinars [at] alzforum [dot] org.
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