Xiongwei Zhu on Accelerating amyloid-beta fibrillization reduces oligomer levels and functional deficits in Alzheimer disease mouse models.
COMMENT Amyloid Spin Doctors It certainly would seem that the Alzheimer disease (AD) research community has completed the 180-degree turnaround on their view of the toxic amyloid-β entity, i.e., from fibrils to oligomers. The days of plaque busters are presumably