Hiroshi Mori on Molecular dissection of domains in mutant presenilin 2 that mediate overproduction of amyloidogenic forms of amyloid beta peptides. Inability of truncated forms of PS2 with familial Alzheimer's disease mutation to increase secretion of Abeta42.
COMMENT It is challengeable and interesting that dimeric formation may be coupled with truncation of PS2. 0 mori