PAPER Wang JZ, Grundke-Iqbal I, Iqbal K
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PAPER Casadesus G, Smith MA, Basu S, Hua J, Capobianco DE, Siedlak SL, Zhu X, Perry G
Increased isoprostane and prostaglandin are prominent in neurons in Alzheimer disease.
Mol Neurodegener. 2007;2:2. PubMed: 17241462PAPER Fischer P, Jungwirth S, Zehetmayer S, Weissgram S, Hoenigschnabl S, Gelpi E, Krampla W, Tragl KH
Conversion from subtypes of mild cognitive impairment to Alzheimer dementia.
Neurology. 2007 Jan 23;68(4):288-91. PubMed: 17242334PAPER Coppedè F, Mancuso M, Lo Gerfo A, Manca ML, Petrozzi L, Migliore L, Siciliano G, Murri L
A Ser326Cys polymorphism in the DNA repair gene hOGG1 is not associated with sporadic Alzheimer's disease.
Neurosci Lett. 2007 Mar 13;414(3):282-5. PubMed: 17240059PAPER Lämsä R, Helisalmi S, Herukka SK, Tapiola T, Pirttilä T, Vepsäläinen S, Hiltunen M, Soininen H
Genetic study evaluating LDLR polymorphisms and Alzheimer's disease.
Neurobiol Aging. 2008 Jun;29(6):848-55. PubMed: 17239995Samir Kumar-Singh on Gain or Loss of Function—Time to Shake up Assumptions on γ-Secretase in Alzheimer Disease?
COMMENT Drs. Davis, De Strooper, and Gabrielle Strobel have done a superb job in taking the initiative to bring this important discussion on our forum. The contributors raised several important points: 1. Dr. Saido noted that not all PS mutations cause FAD. Indee
PAPER Wu YR, Lin HY, Chen CM, Gwinn-Hardy K, Ro LS, Wang YC, Li SH, Hwang JC, Fang K, Hsieh-Li HM, Li ML, Tung LC, Su MT, Lu KT, Lee-Chen GJ
Genetic testing in spinocerebellar ataxia in Taiwan: expansions of trinucleotide repeats in SCA8 and SCA17 are associated with typical Parkinson's disease.
Clin Genet. 2004 Mar;65(3):209-14. PubMed: 14756671Andre Delacourte on Modulation of synaptic plasticity and Tau phosphorylation by wild-type and mutant presenilin1.
COMMENT How is it that PS1(n-/-) mice have on one side severe neurodegeneration (Shen et al., 2007) and here only a subtle impairment in Schaffer LTP? 0 andre.delacourte The presenilin hypothesis of Alzheimer's disease: evidence for a loss-of-function pathog
Thomas Beach on Collective Thought at Its Best: Let’s Contemplate the Centennial
COMMENT These new contributions to the history of Alzheimer disease further illuminate the setting in which Alzheimer made his original observations. I look forward to reading the issue coming out of the Tubingen conference. Many thanks to Andre Delacourte for hi
PAPER Beach TG
The history of Alzheimer's disease: three debates.
J Hist Med Allied Sci. 1987 Jul;42(3):327-49. PubMed: 3305698Bcl-10
ANTIBODY polyclonal manufacturer cat# ab36751 immunogen = recombinant full length protein (human) expressed in E.coli liquid, PBS, BSA, azide, protein A purified Bcl 10 IgG Cell Death Abcam Rabbit Human Bcl-10 http://alzforum.org/res/com/ant/default.asp?antibodyID
Bcl-10 (EP605Y)
ANTIBODY monoclonal EP605Y manufacturer cat# ab40752 immunogen = synthetic peptide corresponding to residues near the N-terminal (human) liquid, tris-glycine saline, azide Bcl-10 <em>not</em> mouse, rat IgG Cell Death Abcam Rabbit Human Bcl-10 http://a
Bcl-6
ANTIBODY polyclonal manufacturer Cat# ab41846 immunogen = synthetic peptide corresp. to internal region of human Bcl6 protein; the aa sequence is unavailable liquid, PBS, BSA, azide, affinity purified Bcl6 others not tested IgG Cell Death Abcam Rabbit Human Bcl-6
Bcl-6
ANTIBODY polyclonal manufacturer Cat# ab41845 immunogen = synthetic peptide corresp. to internal region of human Bcl6 protein liquid, PBS, BSa, azide, protein A purified Bcl6 others not tested IgG Cell Death Abcam Rabbit Human Bcl-6 http://alzforum.org/res/com/ant
Bcl xL
ANTIBODY polyclonal manufacturer cat# ab31396 immunogen = synthetic non-phosphopeptide derived from human Bcl xL around the phosphorylation site of Serine 62 liquid, PBS, glycerol, azide, affinity purified detects endogenous levels of Bcl xL protein around Serine
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