The idea is sound, but neuritic dystrophy means that there is a tauopathy.
For Aβ oligomers that would interfere with LTP, because amyloid deposits are numerous before any significant cognitive impairment, why don't we observe LTP-related defects? Or are Abeta oligomers really toxic in the human brain?
Comments
Inserm
The idea is sound, but neuritic dystrophy means that there is a tauopathy.
For Aβ oligomers that would interfere with LTP, because amyloid deposits are numerous before any significant cognitive impairment, why don't we observe LTP-related defects? Or are Abeta oligomers really toxic in the human brain?
Make a Comment
To make a comment you must login or register.