Delirium Makes its Own Mark on Cognitive Decline
The acute state of confusion not only accelerates memory loss on its own, but also worsens decline caused by other pathologies, including Alzheimer’s disease.
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The acute state of confusion not only accelerates memory loss on its own, but also worsens decline caused by other pathologies, including Alzheimer’s disease.
In induced cells from people with a C9ORF72 expansion, the number of mutant transcripts matched the number of RNA foci per cell.
Neurons of the humble roundworm pack damaged organelles and protein aggregates into giant vesicles and heave them out into extracellular space. Do our neurons do that, too?
Transgenic mouse models devoid of gut microbes make fewer Aβ plaques.
A well-controlled trials shows that a year of treatment offers no cognitive benefit for older men with low testosterone levels.
Produced by the dreaded MRSA bacterium, PSMα3 is the first known example of a natural peptide that forms amyloid fibers made not of β-sheets, but of neatly stacked α-helices.
ALS patients eliminate more p75 neurotrophin receptor than do healthy controls. P75 urine levels rise as disease worsens.
NIH funds a five-year project to validate biomarkers for clinical trials.
Quality control issues may have scuppered clinical trials.
Mnemonic training reshapes functional connectivity patterns in the brain to facilitate recall akin to that found in memory athletes.
The proposed budget would strike a blow to biomedical research, and science in general.
Route to toxicity converges with RNA binding proteins in membrane-less organelles.
A report claims that in the U.K. dementia research grew substantially since 2008, but more gains are needed to find treatments.
Antibodies against tau lower APP expression and degrade Aβ plaques in transgenic mice.
Although shorter tau peptides formed fibrils in a dish, the R3 domain of tau seeded aggregation in cells.