Aβ Fibrils Drive Oligomer Formation, New Model Suggests
assemblies “could be quite varied in different diseases,” Dobson said.—Esther Landhuis None
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assemblies “could be quite varied in different diseases,” Dobson said.—Esther Landhuis None
and pathways.—Esther Landhuis None http://www.alzforum.org/new/detail.asp?id=3497 Alzheimer's Disease Clinical
Disease. Over the coming days, read Esther Landhuis' highlights, which cover new functions for BACE,
Almost half the AD patients had non-convulsive epileptiform activity, Mucke reported.—Esther Landhuis.
levels of inhibition.—Esther Landhuis. This is Part 2 of a three-part series. See also Part 1, Part 3.
to address these issues.—Esther Landhuis. This is Part 1 of a three-part series. See also Part 2, Part 3.
2013). —Esther Landhuis Q&A With Lawrence Steinman. Questions by Esther Landhuis. Q: What motivated
on the ADNI site.—Esther Landhuis. None http://www.alzforum.org/new/detail.asp?id=3448 Alzheimer's
costs of AD by more than 50 percent.”—Esther Landhuis Submitted to The New York Times on 18 March 2013
convinced that what we’re looking at is Aβ,” Ashe said.—Esther Landhuis None
of Pharmaceutical Industries and Associations (EFPIA).—Esther Landhuis None
oligomers influences cognition in this model.—Esther Landhuis None
named winners of the inaugural Breakthrough Prize in Life Sciences (see Science article).—Esther
quality of life in people with neuropathic pain (Boccard et al., 2013).—Esther Landhuis None
related conference story).—Esther Landhuis None http://www.alzforum.org/new/detail.asp?id=3400