Chew the Fat—Lipids, NMDARs Mediate Neuronal Response to Aβ
identify the mechanisms involved.”—Esther Landhuis None http://www.alzforum.org/new/detail.asp?id=2292
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identify the mechanisms involved.”—Esther Landhuis None http://www.alzforum.org/new/detail.asp?id=2292
target is “totally different” from those of other AD drug candidates, Lipton said.—Esther Landhuis None
a combination.”—Esther Landhuis None http://www.alzforum.org/new/detail.asp?id=1923 Stem Cells/Neurogenesis
dysfunction.—Esther Landhuis None http://www.alzforum.org/new/detail.asp?id=2560 Alzheimer's Disease Clinical
contribute to pathogenesis.—Esther Landhuis None http://www.alzforum.org/new/detail.asp?id=1944
receptors is currently used to treat PD patients with dyskinesias.—Esther Landhuis None
neurons sated with lactate (Rouach et al., 2008).—Esther Landhuis None
and Reiman et al., 2009).—Esther Landhuis. This is Part 3 of a four-part series. See Part 1, Part 2,
conclusion is that other genes combine with APP to affect the degeneration of LC neurons.”—Esther Landhuis
Brody. “It raises just as many questions as it answers.”—Esther Landhuis None
Xiaoming Shen of Xinhua Hospital in Shanghai, on the global disease burden analysis.—Esther Landhuis None
underpowered to find association with AD genes.” —Esther Landhuis None
apart, though the relationships are likely to be very complex,” Cirrito noted.—Esther Landhuis None
memory deficits, and then turn it off and see if the memory deficits come back.”—Esther Landhuis None
disease.—Esther Landhuis None http://www.alzforum.org/new/detail.asp?id=2043 Alzheimer's Disease Genetics Aβ