Meynert, Oh, My! Deep Brain Stimulation to Treat Dementia?
of stereotactic NBM stimulation (without STN) in AD patients.—Esther Landhuis None
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of stereotactic NBM stimulation (without STN) in AD patients.—Esther Landhuis None
winded by this level of exercise.”—Esther Landhuis None http://www.alzforum.org/new/detail.asp?id=2685
in the brain in AD and related diseases. For more on that, see Part 3 of this series.—Esther Landhuis.
for high-throughput screening for genes or small molecules that eliminate or modify the pathology.—Esther Landhuis
Longitudinal studies are underway to see if this hypothesis bears out.—Esther Landhuis None
with blocked lysosomal proteolysis.—Esther Landhuis None http://www.alzforum.org/new/detail.asp?id=2803
to tubulin, were able to block QDAβ aggregation.—Esther Landhuis None
event that, if sustained, seems to add to the AD risk conferred by the ApoE4 gene.—Esther Landhuis. None
technology is improving incredibly fast.”—Esther Landhuis None http://www.alzforum.org/new/detail.asp?id=1945
that of mature granule cells,” the authors write.—Esther Landhuis None
toxicity in primary neuronal cultures in a dose-dependent fashion.—Esther Landhuis. This concludes our
of other AD drug candidates, Lipton said.—Esther Landhuis None
wrote in an accompanying editorial.—Esther Landhuis None http://www.alzforum.org/new/detail.asp?id=2569
wrote.—Esther Landhuis None http://www.alzforum.org/new/detail.asp?id=2053 Alzheimer's Disease Biomarkers
variants in the larger genomewide screens, the authors say.—Esther Landhuis None