HAI Seattle: Aβ May Disrupt Brain Function in Normal Seniors
pick up subtle cognitive deficits that escape standard measures.—Esther Landhuis. This is Part 2 of
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pick up subtle cognitive deficits that escape standard measures.—Esther Landhuis. This is Part 2 of
relative utility of various AD biomarker approaches. Stay tuned for the skinny on that.—Esther Landhuis.
next month at the ICAD meeting.—Esther Landhuis. None http://www.alzforum.org/new/detail.asp?id=1852
received information about their ApoE gene status (see ARF Live Discussion).—Esther Landhuis. None
dysregulated gene-jumping frequencies compared to healthy people.—Esther Landhuis None
underlie the onset age heterogeneity (Nelson et al., 2010).—Esther Landhuis None
Dementia Treatment Study Group. To learn all about that, see Part 2 and Part 3.—Esther Landhuis. This is
May 24.—Esther Landhuis None http://www.alzforum.org/new/detail.asp?id=2794 Alzheimer's Disease
similar results as the current genetic knockout approach.—Esther Landhuis None
brain scans, he said. “It may be possible to noninvasively follow the progression of CAA.”—Esther
developments, read Part 2.—Esther Landhuis. This is Part 1 of a four-part series. See also Part 2, Part 3, Part
long-term memory.—Esther Landhuis This is Part 2 of a three-part series. See also Part 1 and Part 3.
hypothesis of AD’ are much more closely related than it initially appeared.”—Esther Landhuis None
roles in lysosomal degradative functions relevant to AD pathogenesis,” he wrote.—Esther Landhuis None
author of an in-press review on omega-3 fatty acids and dementia (Cole et al., 2009).—Esther Landhuis