AD Therapeutic Approaches Tap Complement, Mitochondrial Antioxidant
memory deficits, and then turn it off and see if the memory deficits come back.”—Esther Landhuis None
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memory deficits, and then turn it off and see if the memory deficits come back.”—Esther Landhuis None
to see who develops cognitive impairment.”—Esther Landhuis None
with this reporter.—Esther Landhuis None http://www.alzforum.org/new/detail.asp?id=2193
write.—Esther Landhuis None http://www.alzforum.org/new/detail.asp?id=2191 Cognitive Measures Stem
ARF.—Esther Landhuis None http://www.alzforum.org/new/detail.asp?id=2187 Alzheimer's Disease Aging Immune
to determine which brain areas may be involved in the visual memory processing.—Esther Landhuis None
roles in lysosomal degradative functions relevant to AD pathogenesis,” he wrote.—Esther Landhuis None
author of an in-press review on omega-3 fatty acids and dementia (Cole et al., 2009).—Esther Landhuis
receive the environmental manipulation.—Esther Landhuis None
compartmentalization most likely to control the untoward effects of making cross β-sheet structures.”—Esther Landhuis
stimulation (without STN) in AD patients.—Esther Landhuis None http://www.alzforum.org/new/detail.asp?id=2169
an experimental AD drug on Aβ production (Bateman et al., 2009 and ARF related news story).—Esther
14-16 October in Chicago, immediately preceding this year’s Society for Neuroscience conference.—Esther
in neurons upon NMDA receptor stimulation (Marambaud et al., 2003).—Esther Landhuis None
for the tau splicing regulatory element.—Esther Landhuis None