All Comments by Suzanne Guenette
- The APP Intracellular Domain Is Required for Normal Synaptic Morphology, Synaptic Plasticity, and Hippocampus-Dependent Behavior.
- Novel GαS-protein signaling associated with membrane-tethered amyloid precursor protein intracellular domain.
- Soluble amyloid precursor protein induces rapid neural differentiation of human embryonic stem cells.
- Genetic dissection of the amyloid precursor protein in developmental function and amyloid pathogenesis.
- Keystone: Death Receptor Ligand—New Role for APP, New Model for AD? PAPER RETRACTED.
- The in vivo brain interactome of the amyloid precursor protein.
- The secreted beta-amyloid precursor protein ectodomain APPs alpha is sufficient to rescue the anatomical, behavioral, and electrophysiological abnormalities of APP-deficient mice.
- Involvement of beta-site APP cleaving enzyme 1 (BACE1) in amyloid precursor protein-mediated enhancement of memory and activity-dependent synaptic plasticity.
- Essential roles for Fe65, Alzheimer amyloid precursor-binding protein, in the cellular response to DNA damage.