All Comments by Cheryl Wellington
- Do APOE4’s Lipid Shenanigans Trigger Tauopathy?
- Brain Endothelial Cells Are Diverse, Perturbed by Amyloid
- Human Blood-Brain Barrier Model Blames Pericytes for CAA
- Even Without Amyloid, ApoE4 Weakens Blood-Brain Barrier, Cognition
- 'Organ on a Chip' Models the Ins and Outs of the Blood-Brain Barrier
- Human ApoE Antibody Nips Mouse Amyloid in the Bud
- New Therapeutic Idol? Silencing Ligase Prevents Aβ Deposition in Mice
- Loss-of-function variants in ABCA7 confer risk of Alzheimer's disease.
- Gene transfer of human Apoe isoforms results in differential modulation of amyloid deposition and neurotoxicity in mouse brain.
- ApoE4 Promotes Aβ Oligomerization
- Mechanism of ATP-binding cassette transporter A1-mediated cellular lipid efflux to apolipoprotein A-I and formation of high density lipoprotein particles.
- ABCA1 Loss Lowers ApoE, Not Amyloid; New ApoE Immunology