All Comments by Hiroshi Mori
- The 118-135 peptide of the human prion protein forms amyloid fibrils and induces liposome fusion.
- Sialylated human apolipoprotein E (apoEs) is preferentially associated with neuron-enriched cultures from APOE transgenic mice.
- Estrogen replacement therapy and longitudinal decline in visual memory. A possible protective effect?
- Fibrilization in mouse senile amyloidosis is fibril conformation-dependent.
- Alteration of proteins regulating apoptosis, Bcl-2, Bcl-x, Bax, Bak, Bad, ICH-1 and CPP32, in Alzheimer's disease.
- Metabolism of presenilin 1: influence of presenilin 1 on amyloid precursor protein processing.
- Subcellular distribution and turnover of presenilins in transfected cells.
- Complete genomic screen in late-onset familial Alzheimer's disease.
- Utility of the apolipoprotein E genotype in the diagnosis of Alzheimer's disease. Alzheimer's Disease Centers Consortium on Apolipoprotein E and Alzheimer's Disease.
- No man alone: the rediscovery of Alois Alzheimer's original cases.
- Modulation of beta-amyloid precursor protein processing by the low density lipoprotein receptor-related protein (LRP). Evidence that LRP contributes to the pathogenesis of Alzheimer's disease.
- Copper-binding amyloid precursor protein undergoes a site-specific fragmentation in the reduction of hydrogen peroxide.
- Analysis of message expression in single neurons of Alzheimer's disease brain.
- Presenilin 1 facilitates the constitutive turnover of beta-catenin: differential activity of Alzheimer's disease-linked PS1 mutants in the beta-catenin-signaling pathway.
- Bcl-2 and Bax protein expression in Alzheimer's disease.