PAPER Jesse S, Brettschneider J, Süssmuth SD, Landwehrmeyer BG, von Arnim CA, Ludolph AC, Tumani H, Otto M
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330921 RESULTS
PAPER Li G, Chang M, Jiang H, Xie H, Dong Z, Hu L
Proteomics analysis of methylglyoxal-induced neurotoxic effects in SH-SY5Y cells.
Cell Biochem Funct. 2010 Dec 27; PubMed: 21188731PAPER Kaiser G, Unger A, Marquart B, Weiss M, Freidl M, Wancata J
[The German language version of the Camberwell Assessment of Need for the Elderly (CANE) among dementia patients].
Neuropsychiatr. 2010;24(3):182-9. PubMed: 20926056PAPER
Deep brain stimulation shows promise for Alzheimer's, depression treatment. Doctors also now have more options in how DBS is used to treat Parkinson's disease.
Nat Chem Biol. 2011 Feb;7(2):106-12. PubMed: 21186495PAPER
Can diabetes affect brain function? Does it increase the risk of Alzheimer's disease?
J Neurol. 2011 Jun;258(6):1034-41. PubMed: 21188844Carol Horntrop
Seton Family of HospitalsAustin, United States
PAPER Steiner H, Romig H, Pesold B, Philipp U, Baader M, Citron M, Loetscher H, Jacobsen H, Haass C
Amyloidogenic function of the Alzheimer's disease-associated presenilin 1 in the absence of endoproteolysis.
Biochemistry. 1999 Nov 2;38(44):14600-5. PubMed: 10545183Gal Bitan on Aromatic small molecules remodel toxic soluble oligomers of amyloid beta through three independent pathways.
COMMENT This paper addresses the question of how inhibitors of Aβ-induced toxicity work, and how they affect Aβ self-assembly. This is an important and timely question. In the past, when Aβ fibrils were thought to be the cause of Alzheimer’s disease (AD), inhibit
Aida Attar
University of California, Los AngelesLos Angeles, United States
Huiyuan Li
University of California, Los AngelesLos Angeles, United States
Gal Bitan
University of California, Los AngelesLos Angeles, United States
Paul Coleman on The pathological process underlying Alzheimer's disease in individuals under thirty.
COMMENT This is a vitally important paper that provides further evidence for a "silent period" lasting many decades before AD is clinically diagnosed. The authors found abnormally hyperphosphorylated tau in subcortical regions in people between ages fou
SIRT1/Sir2
ANTIBODY polyclonal manufacturer Cat# 07-131 Immunogen = His-tagged fusion protein corresponding to amino acids 1-131 of mouse Sir2α liquid, purified, Tris-Glycine, saline, azide, glycerol recognizes SIRT1, ~110 kDa IgG Sirtuins Chemicon and Upstate are Millipore
acetylated Tau /ac-tau
ANTIBODY polyclonal laboratory 9AB<br /><a href="http://www.gladstone.ucsf.edu/gladstone/site/gan/" target="_new">Li Gan</a>, Gladstone Institute of Neurological Disease Assistant Professor of Neurology University of Californi
acetylated Tau /ac-tau
ANTIBODY polyclonal laboratory Ab708<br /><a href="http://www.gladstone.ucsf.edu/gladstone/site/gan/" target="_new">Li Gan</a>, Gladstone Institute of Neurological Disease Assistant Professor of Neurology University of Califor
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