331008 RESULTS
COMMENT I am a patient with early Alzheimer's disease who thinks of myself as a "survivor," now six years since diagnosis, having cobbled together a lifestyle regimen of risk reduction strategies with the hope that it will slow the progression of m
COMMENT Advocacy organizations are establishing and managing registries and biobanks are playing a vital role in research. Genetic Alliance has an advocacy-initiated and advocacy-run registry and biobank (Genetic Alliance Registry & BioBank). This is a distin
PAPER Obbard DJ, Gordon KH, Buck AH, Jiggins FM
PAPER Seth RB, Sun L, Chen ZJ
COMMENT The confirmation of Aβ phosphorylation in AD brains in this paper and the suggested contribution to AD pathology supports a role for phosphorylated Aβ. After noting that phosphorylation defects were a major pathological feature of AD, I discovered that Aβ
PAPER Garcia O, Torres M, Helguera P, Coskun P, Busciglio J
PAPER Haier RJ, Head K, Head E, Lott IT
COMMENT In a comment to a recent Alzforum Webinar (1), I outlined a novel, unifying scenario on the etiology of neurodegenerative disorders proposing that APP/Aβ, α-synuclein, tau, huntingtin, TDP-43, prion protein, and other proteins implicated in these disorder
COMMENT We recently suggested that dementia-related hyperhomocysteinemia and its attendant hypomethylation reflect B vitamin depletion due to predictable consequences of neuroinflammatory oxidative stress (1). Framed within the context of an age-based hypothesis,
United States
PAPER Piguet O
PAPER Knopman DS, Roberts RO
PAPER Fidalgo AR, Cibelli M, White JP, Nagy I, Maze M, Ma D
PAPER Cayzac S, Delcasso S, Paz V, Jeantet Y, Cho YH
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