CONFERENCE 2016-10-13 00:00:00-2016-10-14 00:00:00 Glasgow, Scotland 13 – 14 October 2016 The National Task Group on Intellectual Disability and Dementia Practices and the University of Illinois at Chicago, the Scotland at the University of the West of Scotland (UWS), and Alzheimer's ...
COMMENT It is fascinating to follow this debate between the teams of De Strooper/Chávez-Gutiérrez and Shen/Kelleher. The technical arguments presented by both teams in the discussion sections of their recent papers in Neuron are valuable information. However, ...
COMMENT I am very interested in the debate on the pathological effects of PS1 inactive mutations. I rather agree that we should not underestimate the potential role of Aβ43 in the formation of Aβ aggregates. Aβ43 possibly acts as a seed to recruit other Aβ ...
COMMENT The present dispute concerns discrepancies between our finding that FAD-linked Psen1 knock-in (KI) mutations cause a loss of Aβ43 production in the adult mouse brain when mutant PS1 is expressed at normal levels and with the normal spatiotemporal pattern, ...
PAPER Huijbers MG, Niks EH, Klooster R, de Visser M, Kuks JB, Veldink JH, Klarenbeek P, Van Damme P, de Baets MH, van der Maarel SM, van den Berg LH, Verschuuren JJ27133662
PAPER Staff NP, Appel SH27133660
PAPER Moulin S, Labreuche J, Bombois S, Rossi C, Boulouis G, Hénon H, Duhamel A, Leys D, Cordonnier C27133238
PAPER Hasegawa M
Molecular Mechanisms in the Pathogenesis of Alzheimer's disease and Tauopathies-Prion-Like Seeded Aggregation and Phosphorylation.Biomolecules. 2016;6(2) PubMed: 27136595
PAPER Gupta VK, Chitranshi N, Gupta VB, Golzan M, Dheer Y, Wall RV, Georgevsky D, King AE, Vickers JC, Chung R, Graham S
Amyloid β accumulation and Inner retinal degenerative changes in Alzheimer's disease transgenic mouse.Neurosci Lett. 2016 Apr 28; PubMed: 27133194
PAPER Favaretto S, Walter U, Baracchini C, Pompanin S, Bussè C, Zorzi G, Ermani M, Cagnin A27132982
PAPER Rosenberg RN, Lambracht-Washington D, Yu G, Xia W27135718
RESEARCH NEWS 2016-05-03 Research News Repeat expansions in the C9ORF72 gene increase risk for amyotrophic lateral sclerosis and frontotemporal dementia, but scientists have struggled to develop a mouse model that recapitulates the full spectrum of disease symptoms. In the April ...
Ludwig-Maximilians-University Munich and German Center for Neurodegenerative Diseases, MunichGermany
University of BordeauxBordeaux, France
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