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Home: Research: Forums: Live Discussions

What We Know, What We Don't Know

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	What we know	What we don’t know
17.	In ApoE4 carriers, and perhaps also in eFAD mutation carriers, activation in medial temporal regions is increased decades before expected onset of any AD	What accounts for increased activation? Is this related in any way to the increased risk of AD?

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			For the purpose of designing intervention trials for people with FAD mutation, is a presymptomatic individual a:

				Healthy person? (Drug trial risk tolerance: zero)

				Ill person? (Risk tolerance: medium)

				Person with fatal disease? (Risk tolerance: high, just check cancer.)








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