. Transgenic mice overexpressing amyloid beta protein are an incomplete model of Alzheimer disease. Exp Neurol. 2004 Jul;188(1):52-64. PubMed.

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  1. The title of this paper should, apparently really be "Transgenic mice overexpressing amyloid beta protein ALONE are an incomplete ----". When expression of mutated APP is combined with expression of other mutated proteins important in AD pathology these t.g. mice exhibit a rich array of AD characteristics (e.g. LaFerla, 2003).

    References:

    . Amyloid deposition precedes tangle formation in a triple transgenic model of Alzheimer's disease. Neurobiol Aging. 2003 Dec;24(8):1063-70. PubMed.

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