. Mutation of the Phe20 residue in Alzheimer's amyloid beta-peptide might decrease its toxicity due to disruption of the Met35-cupric site electron transfer pathway. Chem Res Toxicol. 2004 Mar;17(3):325-9. PubMed.

Recommends

Please login to recommend the paper.

Comments

Comments on this Paper

No Available Comments

Make a Comment

To make a comment you must login or register.

This paper appears in the following:

Webinars

  1. Role and Control of Metal-Mediated Fibril Toxicity