This paper appears in the following:
- Keystone: Partners in Crime—Do Aβ and Prion Protein Pummel Plasticity?
- Amyloid and Prions—Co-Conspirators in Disease?
- Model Shows Oligomers Impair Memory, Questions Role of Prion Protein
- Neuronal Glutamate Fuels Aβ-induced LTD
- Shape-shifting Prions: Infectious Recombinant and Myelin-Minding Normal
- Patient Aβ Dimers Sufficient for Tau, Neuritic Changes
- Patient-Derived Aβ Needs Prion Protein to Harm Synapses