E2F1 mediates death of B-amyloid-treated cortical neurons in a manner independent of p53 and dependent on Bax and caspase 3. - AlzForum Alzheimer Research Paper of the Week


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Home: Papers of the Week

Annotation

	Giovanni A, Keramaris E, Morris EJ, Hou ST, O'Hare M, Dyson N, Robertson GS, Slack RS, Park DS. E2F1 mediates death of B-amyloid-treated cortical neurons in a manner independent of p53 and dependent on Bax and caspase 3. J Biol Chem. 2000 Apr 21;275(16):11553-60. PubMed Abstract

	Comments on Paper and Primary News

	Comment by: Hiroshi Mori, ARF Advisor
	Permalink
	This is one of the interesting papers in AD. Several lines of knowledge on molecules such as p53 or other caspases involved in the well-known cellular oncogenic or pathological cascades are welcome in AD-related cell death because of simple interest and for the background information this brings to the AD field. View all comments by Hiroshi Mori

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