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Tau protein formation is part of the cascade of events that occurs after aluminosilicate particulates are formed. The initiating event is the precipitation of aluminosilicate crystals.  View all comments by Colin Meyer |
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The above poll questions represent the amalgam of tau in AD. Picking just one of them does not do justice to tau. The last few years gave convincing evidence that tau follows Aβ, that tau pathology is a typical consequence of neurodegeneration, and that disturbed tau function eliminates critical neuronal function. However, to fully answer the question of what's tau got to do with AD, one first would need to know the mechanism and consequences of Aβ foul play in AD. Knowing that, Aβ may turn tau into the main malefactor for second-level neurodegeneration, or tau may just be a casual bystander caught in the crowd of neuronal damage. View all comments by Tobias Hartmann |
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