Non-Motor Symptoms Are an Early Sign of Parkinson's
A new study supports the idea that non-motor symptoms of Parkinson's disease may aid earlier diagnosis.
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A new study supports the idea that non-motor symptoms of Parkinson's disease may aid earlier diagnosis.
Amyloid fibrils seeded by Alzheimer’s brain extracts indicate that Aβ forms specific strains in different individuals.
A deletion in chromosome 22 points to new genetic risk factors for Parkinson's disease.
FUS, a protein tied to neurodegeneration, normally mends DNA breaks, according to a paper in Nature Neuroscience.
It's not just for neurodevelopment anymore. Death receptor 6—known for pruning axons—also trims unused connections in adulthood, hinting at a function in Alzheimer's.
The NIH announced $45 million in new funding to support trials in preclinical Alzheimer’s populations, as well as efforts to identify new therapeutic targets.
New research proposes a neuroimmune receptor as the latest Aβ binding partner.
The first report on PBB3, the latest tau ligand for brain imaging under development, suggests it binds all types of tau aggregate.
Proposed preclinical stages for Alzheimer’s disease predict well who is most likely to progress to Alzheimer’s dementia.
What’s better for future treatment of Parkinson’s: patient-derived or banked iPSCs? In comparison, patient’s own come out on top.
Antibodies against extracellular tau block seeding, diminish brain pathology, and may improve cognition in a mouse model.
A new RNA interference therapy for amyotrophic lateral sclerosis has some success in mice and monkeys.
Advances in RNA sequencing are shedding light on gene regulation and helping scientists make sense of data from genome-wide association studies.
Rare mutations in the ADAM10 gene make a genetic case for the amyloid cascade as a cause of late onset Alzheimer's disease.
In the first study of its kind, researchers found a surprisingly low incidence of Lewy body dementias in a U.S. population.
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