Bridges and Blues—Striking a Chord for Axon Protection
Scientists continue to sound out new strategies for re-establishing broken neural connections and for protecting injured spinal cords from further damage...
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Scientists continue to sound out new strategies for re-establishing broken neural connections and for protecting injured spinal cords from further damage...
Presenilin mutations cause familial Alzheimer disease, but just how they do their damage is not understood...
Uppsala: Immunotherapy and Biomarkers Take Center Stage Uppsala: Brainstorming Biomarkers Uppsala University, Sweden, played host to “Neurodegenerative Disorders—Immunotherapy and Biomarkers,” an international conference held 28-29 May 2009, and organized
A variety of approaches are beginning to home in on Aβ oligomers as drug targets, and some of them were showcased at the International Conference on Alzheimer’s Disease...
Whether Aβ amyloid portends future dementia isn’t so clear, but a report in the July 30 Neuron puts meat and bones on the hunch that it does...
Biomarkers were top of the agenda on the second day of Neurodegenerative Disorders—Immunotherapy and Biomarkers...
The ICAD featured that the interest in small Aβ aggregates has branched out far beyond the initial debates about whether they even exist and which forms are important...
The motor neurons and surrounding cells damaged in ALS cry out for assistance, and scientists are developing ways to home in on that need with growth factors that slow the degeneration process...
Anesthesia is meant to induce a temporary fog, lifting as soon as the drugs wear off. But for some, the effects linger, with delirium or confusion for days or weeks following surgery...
This story presents an update from this year's ICAD conference, together with an extended Q&A with the developer of RemberTM, Claude Wischik...
At the ICAD, news on the drug trials landscape painted a mostly bleak picture, but of course, that’s not the whole story...
A new paper shows that stem cells are able to rehabilitate memory function in old mice with extensive AD pathology, but not by replacing lost neurons...
The endoplasmic reticulum protein reticulon-3 (RTN3) can prevent amyloid-β plaques—but only if it doesn’t become aggregated itself...
On the last morning of the International Conference on Alzheimer’s Disease (ICAD), investigators from different research groups marked a milestone in AD genetics...
On the heels of a report that tauopathy can be spread throughout the brain by the injection of a small amount of toxic tau comes a similar paper on the propagation of (Aβ)...