Results raise the tentative prospect that a medical food might have possible cognitive benefit early in disease.
Tracer uptake in the brain matches autopsy data. This opens up new possibilities for in vivo disease staging and longitudinal studies.
By modifying a cation channel to respond to magnetic fields, scientists can now control neural activity quickly and noninvasively.
New online venues welcome data that tend to get the cold shoulder from traditional journals. They offer open peer review and the ability to link papers. Nice try or publishing revolution?
Multiple studies link the amount and processing of this cell surface protein to inflammation and neurodegeneration, although what it does remains unclear.
Scientists wonder if the sodium channel blocker might also slow disease progression.
Scientists correlate increased appetite and sugar cravings in frontotemporal dementia patients with localized atrophy in the brain.
Healthy neurons tell astrocytes how to act. In the case of ALS, astrocyte signals spell trouble.
Exercising the brain and body in middle age may not limit progression of Alzheimer’s pathology.
Proteolysis of other substrates, including Neuregulin, proceeds apace.
An Italian study links the supposedly protective ApoE2 gene to frontotemporal dementia in amyotrophic lateral sclerosis.
Researchers report the first successful use of this technology to detect soluble Aβ in the brains of Alzheimer’s mouse models.
Genetic deletion of white blood cells in an Alzheimer’s mouse model quadrupled plaque load, but restoring peripheral antibodies halved this increase.
Overly active interneurons fire up pyramidal neurons in mice with mutant TDP-43.
A new procedure turns circulating immune cells into neurons. It could simplify how scientists make patient-specific disease models.
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