Experimental AD Vaccine Hitches a Ride on Tetanus Toxin
A new AD vaccine co-opts memory T helper cells to cajole B cells into producing anti-Aβ antibodies...
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A new AD vaccine co-opts memory T helper cells to cajole B cells into producing anti-Aβ antibodies...
Scientists have turned their attention toward testing therapies in early-stage patients who have underlying brain pathology but little to no functional impairment...
If another small, hopeful trial is to be believed, pooled antibody treatment has the potential to stave off dementia...
Neurofibrillary tangles of tau protein litter Alzheimer’s disease brains, but are they actually bad for neurons?...
DNA damage accrues in Alzheimer's disease and in the aging brain, but would anyone have predicted that neural activity is to blame?...
After more than a decade of seemingly pushing against a wall, BACE inhibitors for Alzheimer’s disease are finally surging forward...
Adding insult to injury, many patients with a movement disorder get dementia, too...
Microglia Activation—Venusberg Meeting Questions M1, M2 Designations Inflammatory Crosstalk Between Periphery and Brain Blessing or Curse? Peripheral Cytokines in the Brain Glial Imaging—Amid Slow Progress, EU Project Takes Up Challenge Venusberg Meeting
Some dismiss it as a therapeutic dead end, but for others, the M1 muscarinic receptor is a target for ongoing drug development research in Alzheimer’s disease...
Calls for functional markers emanated loud and clear from the 3rd Venusberg Meeting on Neuroinflammation...
A cancer drug newly proposed for neurodegenerative diseases seems to do it all...
With variants that boost risk for both familial and sporadic Parkinson’s disease, the LRRK2 gene beckons as a prime target for therapy development...
The subject of crosstalk between the peripheral circulation and the central nervous system permeated conversation...
As magnetic resonance imaging (MRI) resolution improves and more animal models become available, scientists are stepping up their use of mouse MRI...
Pyroglutamate Aβ is a particularly toxic version of the peptide at the center of AD pathogenesis; in fact, some scientists claim it seeds the formation of plaques...