All Comments by Vincent Marchesi

  1. Amyloid precursor protein overexpression depresses excitatory transmission through both presynaptic and postsynaptic mechanisms.
  2. AMPA Receptors: Going, Going, Gone in Aβ-exposed Synapses, PSD95 Knockouts
  3. Gain or Loss of Function—Time to Shake up Assumptions on γ-Secretase in Alzheimer Disease?
  4. Aβ Star is Born? Memory Loss in APP Mice Blamed on Oligomer
  5. An alternative interpretation of the amyloid Abeta hypothesis with regard to the pathogenesis of Alzheimer's disease.
  6. Messing with the Membrane—An Alternative Interpretation of the Amyloid-β Hypothesis
  7. An alternative interpretation of the amyloid Abeta hypothesis with regard to the pathogenesis of Alzheimer's disease.
  8. Messing with the Membrane—An Alternative Interpretation of the Amyloid-β Hypothesis
  9. An alternative interpretation of the amyloid Abeta hypothesis with regard to the pathogenesis of Alzheimer's disease.
  10. Messing with the Membrane—An Alternative Interpretation of the Amyloid-β Hypothesis
  11. Virosome-based active immunization targets soluble amyloid species rather than plaques in a transgenic mouse model of Alzheimer's disease.
  12. An alternative interpretation of the amyloid Abeta hypothesis with regard to the pathogenesis of Alzheimer's disease.
  13. Messing with the Membrane—An Alternative Interpretation of the Amyloid-β Hypothesis
  14. Nicastrin—A DAPper Role in γ-Secretase
  15. Amyloid Hypothesis—Closing the Spigot Helps, Temporarily