All Comments by Nikolaos K. Robakis
- Society for Neuroscience Annual Meeting: Presenilins: An Overview
- Somatic mutation analysis of the APP and Presenilin 1 and 2 genes in Alzheimer's disease brains.
- Modification of tau to an Alzheimer's type protein interferes with its interaction with microtubules.
- Immunologic treatment of Alzheimer's disease.
- A familial case of Alzheimer's disease without tau pathology may be linked with chromosome 3 markers.
- Mutation-specific functional impairments in distinct tau isoforms of hereditary FTDP-17.
- Soluble pool of Abeta amyloid as a determinant of severity of neurodegeneration in Alzheimer's disease.
- Neuronal overexpression of mutant amyloid precursor protein results in prominent deposition of cerebrovascular amyloid.
- Evidence that intramolecular associations between presenilin domains are obligatory for endoproteolytic processing.
- Two amyloid precursor protein transgenic mouse models with Alzheimer disease-like pathology.
- Inhibition of platelet activation by the Alzheimer's disease amyloid precursor protein.
- alpha-Synuclein in filamentous inclusions of Lewy bodies from Parkinson's disease and dementia with lewy bodies.
- Interaction of Alzheimer beta-amyloid peptide(1-40) with lipid membranes.
- Accumulation of SNAP-25 immunoreactive material in axons of Alzheimer's disease.
- A protein kinase, PKN, accumulates in Alzheimer neurofibrillary tangles and associated endoplasmic reticulum-derived vesicles and phosphorylates tau protein.