All Comments by Jürgen Götz
- One at a time, live tracking of NGF axonal transport using quantum dots.
- Multiple proteins implicated in neurodegenerative diseases accumulate in axons after brain trauma in humans.
- Mechanisms of impaired mitochondrial energy metabolism in acute and chronic neurodegenerative disorders.
- Increased autophagic degradation of mitochondria in Alzheimer disease.
- Soluble oligomers from a non-disease related protein mimic Abeta-induced tau hyperphosphorylation and neurodegeneration.
- Single molecule profiling of tau gene expression in Alzheimer's disease.
- Somatodendritic microRNAs identified by laser capture and multiplex RT-PCR.
- The genetics of frontotemporal lobar degeneration.
- Tauopathy models and human neuropathology: similarities and differences.
- Annonacin, a natural mitochondrial complex I inhibitor, causes tau pathology in cultured neurons.
- Mechanisms of mitochondrial dysfunction and energy deficiency in Alzheimer's disease.
- AGEing Neurons Waste Away in Fly Model of Neurodegeneration
- Mutant Tau Sharpens Wits of Young Mice
- Is Motor Neuron Axonopathy behind Movement Problems in Alzheimer Disease?
- Dangerous Liaisons—Tau and Aβ, Together at Last?