All Comments by Paul Coleman

  1. Absence of aluminium in neurofibrillary tangles in Alzheimer's disease.
  2. cAMP-dependent protein kinase phosphorylations on tau in Alzheimer's disease.
  3. Environmental enrichment inhibits spontaneous apoptosis, prevents seizures and is neuroprotective.
  4. Elevated expression of p21ras is an early event in Alzheimer's disease and precedes neurofibrillary degeneration.
  5. Occupational exposures to solvents and aluminium and estimated risk of Alzheimer's disease.
  6. Cytoarchitectonic alterations in the supramarginal gyrus of late onset Alzheimer's disease.
  7. Dephosphorylation of tau during transient forebrain ischemia in the rat.
  8. Striation is the characteristic neuritic abnormality in Alzheimer disease.
  9. Age-related alteration of PKC, a key enzyme in memory processes: physiological and pathological examples.
  10. Vulnerability of small GABAergic neurons to human beta-amyloid pentapeptide.
  11. Free D- and L-amino acids in ventricular cerebrospinal fluid from Alzheimer and normal subjects.
  12. Aluminum but not iron treatment induces pro-oxidant events in the rat brain.
  13. A quantitative study of neurofibrillary tangles, senile plaques and astrocytes in the hippocampal subdivisions and entorhinal cortex in Alzheimer's disease, normal controls and non-Alzheimer neuropsychiatric diseases.
  14. Link Between Education Level and AD Risk Strengthened