All Comments by Ilya Bezprozvanny
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- Whole Genome Analysis Nets New Lewy Body Dementia Gene
- And Then There Were Three: Donanemab Phase 3 Trial Positive
- RIKEN Says Its Mouse Models Are Now Easier to Obtain
- Sylvain Lesné, Who Found Aβ*56, Accused of Image Manipulation
- Peering Inside Stubby and Mushroom Dendritic Spines
- Mutant Presenilin Skews Calcium Homeostasis by Chomping on ER Sensor
- Shielding Synaptic Glutamate Receptor from Aβ Preserves Memory in Mice
- Reversal of Aging-Related Neuronal Ca2+ Dysregulation and Cognitive Impairment by Delivery of a Transgene Encoding FK506-Binding Protein 12.6/1b to the Hippocampus.
- Presenilin-1 knockin mice reveal loss-of-function mechanism for familial Alzheimer's disease.
- ER-mitochondria associations are regulated by the VAPB-PTPIP51 interaction and are disrupted by ALS/FTD-associated TDP-43.
- Cytotoxicity of intracellular aβ42 amyloid oligomers involves Ca2+ release from the endoplasmic reticulum by stimulated production of inositol trisphosphate.
- First Crystal Structure of a Presenilin
- Early presynaptic and postsynaptic calcium signaling abnormalities mask underlying synaptic depression in presymptomatic Alzheimer's disease mice.
- Single-channel Ca(2+) imaging implicates Aβ1-42 amyloid pores in Alzheimer's disease pathology.
- A synaptic vesicle-associated Ca2+ channel promotes endocytosis and couples exocytosis to endocytosis.