All Comments by Inez Vincent

  1. Apolipoprotein E and Reelin ligands modulate tau phosphorylation through an apolipoprotein E receptor/disabled-1/glycogen synthase kinase-3beta cascade.
  2. Ectopic expression of cell cycle markers in models of induced programmed cell death in dopamine neurons of the rat substantia nigra pars compacta.
  3. Oxidative Stress Triggers Neuronal Cell-Cycle Reentry
  4. p53 as Mediator of Oxidative Stress and Inflammation?
  5. Cyclin D1 and cyclin E are co-localized with cyclo-oxygenase 2 (COX-2) in pyramidal neurons in Alzheimer disease temporal cortex.
  6. Nuclear localization of N-terminal mutant huntingtin is cell cycle dependent.
  7. p35/Cdk5 pathway mediates soluble amyloid-beta peptide-induced tau phosphorylation in vitro.
  8. p35/Cdk5 pathway mediates soluble amyloid-beta peptide-induced tau phosphorylation in vitro.
  9. Prominent neurodegeneration and increased plaque formation in complement-inhibited Alzheimer's mice.
  10. Cyclin-dependent kinases and P53 pathways are activated independently and mediate Bax activation in neurons after DNA damage.
  11. Reduced protein phosphatase 2A activity induces hyperphosphorylation and altered compartmentalization of tau in transgenic mice.
  12. Accumulation of C-terminally truncated tau protein associated with vulnerability of the perforant pathway in early stages of neurofibrillary pathology in Alzheimer's disease.
  13. Metabolic, metallic, and mitotic sources of oxidative stress in Alzheimer disease.
  14. Loss and atrophy of layer II entorhinal cortex neurons in elderly people with mild cognitive impairment.
  15. Where do Alzheimer's plaques and tangles come from? Aging-induced protein degradation inefficiency.