All Comments by Hiroshi Mori

  1. Possible role of tau protein kinases in pathogenesis of Alzheimer's disease.
  2. Bleomycin hydrolase is associated with risk of sporadic Alzheimer's disease.
  3. Intracellular processing of aggregated tau differs between corticobasal degeneration and progressive supranuclear palsy.
  4. An immunochemical study on tau glycation in paired helical filaments.
  5. Transferrin C2, metal binding and Alzheimer's disease.
  6. Quantifying amyloid beta-peptide (Abeta) aggregation using the Congo red-Abeta (CR-abeta) spectrophotometric assay.
  7. Opposite roles of apolipoprotein E in normal brains and in Alzheimer's disease.
  8. Neurotrophic factors [activity-dependent neurotrophic factor (ADNF) and basic fibroblast growth factor (bFGF)] interrupt excitotoxic neurodegenerative cascades promoted by a PS1 mutation.
  9. Wide range of disease onset in a family with Alzheimer disease and a His163Tyr mutation in the presenilin-1 gene.
  10. Pathologic amyloid beta-protein cell surface fibril assembly on cultured human cerebrovascular smooth muscle cells.
  11. The effects of aging and neurodegeneration on apoptosis-associated DNA fragmentation and the benefits of nicotinamide.
  12. Fibrilization in mouse senile amyloidosis is fibril conformation-dependent.
  13. Complement C1-inhibitor expression in Alzheimer's disease.
  14. ApoE polymorphism in Polish patients with Alzheimer's disease.
  15. Neurofibrillary degeneration in progressive supranuclear palsy and corticobasal degeneration: tau pathologies with exclusively "exon 10" isoforms.