All Comments by Gunnar Gouras
- Mechanism of amyloid plaque formation suggests an intracellular basis of Abeta pathogenicity.
- Together at Last, Top Five Biomarkers Model Stages of AD
- What’s My Line?—Fishing for the True Role of Aβ
- Characterizing the appearance and growth of amyloid plaques in APP/PS1 mice.
- Popcorn Plaque? Alzheimer Disease Is Slow, Yet Plaque Growth Is Fast
- Internalized antibodies to the Abeta domain of APP reduce neuronal Abeta and protect against synaptic alterations.
- AMPA Receptors: Going, Going, Gone in Aβ-exposed Synapses, PSD95 Knockouts
- Chew ’em Up and Spit ’em Out: Aβ Leaves Cells via Exosomes
- A specific amyloid-beta protein assembly in the brain impairs memory.
- Amyloid Precursor Protein and Notch Intracellular Domains are Generated after Transport of their Precursors to the Cell Surface
- An alternative interpretation of the amyloid Abeta hypothesis with regard to the pathogenesis of Alzheimer's disease.
- Messing with the Membrane—An Alternative Interpretation of the Amyloid-β Hypothesis
- BACE overexpression alters the subcellular processing of APP and inhibits Abeta deposition in vivo.
- San Diego: Treating Forgetfulness—Triple Transgenics Provoke
- Dendrite and dendritic spine alterations in Alzheimer models.