All Comments by Gunnar Gouras

  1. Intraneuronal APP, not free Aβ peptides in 3xTg-AD mice: implications for tau versus Aβ-mediated Alzheimer neurodegeneration.
  2. Do Overactive Brain Networks Broadcast Alzheimer’s Pathology?
  3. Effects of synaptic modulation on beta-amyloid, synaptophysin, and memory performance in Alzheimer's disease transgenic mice.
  4. Deleterious effects of amyloid beta oligomers acting as an extracellular scaffold for mGluR5.
  5. Analysis of microdissected human neurons by a sensitive ELISA reveals a correlation between elevated intracellular concentrations of Abeta42 and Alzheimer's disease neuropathology.
  6. Mechanism of amyloid plaque formation suggests an intracellular basis of Abeta pathogenicity.
  7. Together at Last, Top Five Biomarkers Model Stages of AD
  8. What’s My Line?—Fishing for the True Role of Aβ
  9. Characterizing the appearance and growth of amyloid plaques in APP/PS1 mice.
  10. Popcorn Plaque? Alzheimer Disease Is Slow, Yet Plaque Growth Is Fast
  11. Internalized antibodies to the Abeta domain of APP reduce neuronal Abeta and protect against synaptic alterations.
  12. AMPA Receptors: Going, Going, Gone in Aβ-exposed Synapses, PSD95 Knockouts
  13. Chew ’em Up and Spit ’em Out: Aβ Leaves Cells via Exosomes
  14. A specific amyloid-beta protein assembly in the brain impairs memory.
  15. Amyloid Precursor Protein and Notch Intracellular Domains are Generated after Transport of their Precursors to the Cell Surface