All Comments by Charles Glabe

  1. Nanotools for megaproblems: probing protein misfolding diseases using nanomedicine modus operandi.
  2. Prefibrillar amyloid aggregates could be generic toxins in higher organisms.
  3. Green tea (-)-epigallocatechin-gallate modulates early events in huntingtin misfolding and reduces toxicity in Huntington's disease models.
  4. Amyloid-beta peptide remnants in AN-1792-immunized Alzheimer's disease patients: a biochemical analysis.
  5. Structural differences in Abeta amyloid protofibrils and fibrils mapped by hydrogen exchange--mass spectrometry with on-line proteolytic fragmentation.
  6. Degradation of the amyloid beta-protein by the novel mitochondrial peptidasome, PreP.
  7. Neuroserpin binds Abeta and is a neuroprotective component of amyloid plaques in Alzheimer disease.
  8. Inhibition of APP trafficking by tau protein does not increase the generation of amyloid-beta peptides.
  9. Dendrimeric Abeta1-15 is an effective immunogen in wildtype and APP-tg mice.
  10. Biotin-avidin interaction-based screening assay for Alzheimer's beta-peptide oligomer inhibitors.
  11. Olfaction in neurodegenerative disorder.
  12. SfN: Amyloid Oligomers—Not So Elusive, After All? Part 2
  13. <i>Drosophila</i> APP Signals Synapse Formation
  14. Exacerbation of cerebral amyloid angiopathy-associated microhemorrhage in amyloid precursor protein transgenic mice by immunotherapy is dependent on antibody recognition of deposited forms of amyloid beta.
  15. Conformation Rules Part 2: News, Common Threads, Debate from San Diego Protein Misfolding Conference